Christopher Ingalls
Professor Kinesiology and Health- Education
Ph. D. in Kinesiology, Texas A&M University, 1994
Master of Science in Kinesiology, Texas A&M University, 1990
Bachelor of Science in Health and Sport Science, Wake Forest University,1988
- Specializations
Muscle physiology and biochemistry:
1) Molecular mechanisms of strength loss and recovery associated with exercise-induced skeletal muscle injury,
2) Molecular and cellular mechanisms of skeletal muscle tissue adaptation to exercise, and
3) Impact of exercise-induced skeletal muscle weakness on developing secondary musculoskeletal injuries.
- Biography
Christopher Ingalls has been studying skeletal muscle physiology since 1988. The focus of his research for the last 28 years has been to understand the causes of strength loss and recovery associated with exercise-induced skeletal muscle injury in humans and animal models.
To date, his 43 peer-reviewed research papers have been cited nearly 3,000 times in journal articles and textbooks. His research on skeletal muscle physiology and injury in models of human muscle disease has been funded by the National Institutes of Health for over 10 years. Current research projects are focused on translating the impact of differential skeletal muscle injury on joint stability during locomotion and the subsequent development of musculoskeletal injuries.
- Publications
Corona, B.T. and C.P. Ingalls. (2013). “Immediate force loss after eccentric contractions is increased with L-NAME administration, a nitric oxide synthase inhibitor”. Muscle & Nerve 47(2):271-273.
Rouviere C., B.T. Corona, and C.P. Ingalls. (2012). “Oxidative Capacity and Fatigability in Run Trained Malignant Hyperthermia Susceptible Mice.” Muscle & Nerve 45(4): 586-596.
Corona, B.T, S.L. Hamilton, and C.P. Ingalls. (2010). “The effect of prior exercise on thermal sensitivity of malignant hyperthermia susceptible muscle.” Muscle & Nerve 42(2): 270-272.
Corona, B.T., E.M. Balog, J.A. Doyle, J.C. Rupp, R.C. Luke, and C.P. Ingalls. (2010). “Junctophilin damage contributes to early strength deficits and EC coupling failure after eccentric contractions.” Am. J . Physiol. Cell Physiol. 298: C365-376.
Corona, B, C. Rouviere, S.L. Hamilton, and C.P. Ingalls. (2008). “FKBP12 deficiency reduces strength deficits after eccentric contraction-induced injury.” J. Appl. Physiol. 105: 527-537.